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A CRISPRi/a platform in human iPSC-derived microglia uncovers regulators of disease states
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A signaling molecule called neurotensin shapes whether environmental cues are associated with positive or negative memories in mouse model.
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A large-scale genome-wide cross-trait analysis reveals shared genetic architecture between Alzheimer’s disease and gastrointestinal tract disorders
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Shared genetic architecture between Alzheimer’s disease and gastrointestinal tract disorders
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Whole Blood Exchange Being Studied as a Potential Treatment for Alzheimers Disease
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Mechanism of site-specific tau phosphorylation linked to Alzheimers Disease-related hyperphosphorylation defines master sites that govern phosphorylation across the entire tau molecule
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Psylocybin Improves Mental Health in Microdoses
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Imipramine and olanzapine block apoE4-catalyzed polymerization of A-beta and show evidence of improving Alzheimer’s disease cognition
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Transcorneal electrical stimulation enhances cognitive functions in aged and 5XFAD mouse models
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A pathway that begins in the gut and ends with a potent pro-inflammatory toxin in brain cells contributing to the development of Alzheimers disease
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A-beta 1-40 builds up in the walls of pial arteries (that control the supply of blood and oxygen) and reduces blood flow to the brain, causing memory loss.
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Influenza vaccination associated with a 40% decrease in the 4-year risk of developing Alzheimers Disease in patients 65 years or older.
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An add-on probiotic treatment changes the gut microbiota and ameliorates depressive symptoms
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An organelle may target tau degradation
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Synaptic connections restored in mouse model of Alzheimers when given the drug known as Silent Allosteric Modulation or SAM
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Changes in FMNL2 activity caused by cerebrovascular disease prevent the efficient clearance of toxic proteins from the brain, eventually leading to Alzheimer’s disease
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Vitamin D deficiency associated with neuroimaging outcomes and the risks of dementia and stroke
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A Single Molecule May Result in Alzheimers