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Intermittent fasting protects against Alzheimer’s disease in mice by altering metabolism through remodeling of the gut microbiota
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Single-Dose Psilocybin for a Treatment-Resistant Episode of Major Depression
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Cerebrovascular insulin receptors are defective in Alzheimer’s disease
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Induced neurons represent a platform for examining the molecular mechanisms underlying PTSD, identifying biomarkers of stress response, and conducting drug screening to identify new therapeutics
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Phagocytic clearance of neurotoxic material by microglia is dependent on SYK, a pivotal regulator of microglial activation and AKT/GSK3β-signaling, in mouse model
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The three-dimensional landscape of cortical chromatin accessibility in Alzheimer’s disease
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Fasting-mimicking diet cycles reduce neuroinflammation to attenuate cognitive decline in Alzheimer’s models
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Intranasally delivered oxytocin derivative, PAS-CPPs-oxytocin, increases synaptic plasticity and reverses cognitive impairment in mouse model of Alzheimer’s Disease
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A CRISPRi/a platform in human iPSC-derived microglia uncovers regulators of disease states
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A signaling molecule called neurotensin shapes whether environmental cues are associated with positive or negative memories in mouse model.
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A large-scale genome-wide cross-trait analysis reveals shared genetic architecture between Alzheimer’s disease and gastrointestinal tract disorders
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Shared genetic architecture between Alzheimer’s disease and gastrointestinal tract disorders
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Whole Blood Exchange Being Studied as a Potential Treatment for Alzheimers Disease
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Mechanism of site-specific tau phosphorylation linked to Alzheimers Disease-related hyperphosphorylation defines master sites that govern phosphorylation across the entire tau molecule
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Psylocybin Improves Mental Health in Microdoses
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Imipramine and olanzapine block apoE4-catalyzed polymerization of A-beta and show evidence of improving Alzheimer’s disease cognition
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Transcorneal electrical stimulation enhances cognitive functions in aged and 5XFAD mouse models
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A pathway that begins in the gut and ends with a potent pro-inflammatory toxin in brain cells contributing to the development of Alzheimers disease